Cell-Based Therapy For Chronic Neuropathic Pain After CNS Injury - Hains, Bryan
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Hemisection spinal cord injury (SCI) in preclinical models results in pathological and functional features that mimic the human condition. Chronic pain secondary to neuronal injury is actively and continuously modulated at multiple locations along the sensory neuraxis. Here, we describe how pain-processing neurons of the spinal cord process and communicate nociceptive information in terms of precisely calibrated firing patterns. We then discuss how several cell types, with a focus on 5-HTergic neurons, cause system-wide interference in nociceptive processing through novel signaling schema,…mehr

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Produktbeschreibung
Hemisection spinal cord injury (SCI) in preclinical models results in pathological and functional features that mimic the human condition. Chronic pain secondary to neuronal injury is actively and continuously modulated at multiple locations along the sensory neuraxis. Here, we describe how pain-processing neurons of the spinal cord process and communicate nociceptive information in terms of precisely calibrated firing patterns. We then discuss how several cell types, with a focus on 5-HTergic neurons, cause system-wide interference in nociceptive processing through novel signaling schema, thus contributing to sensory network plasticity and chronic pain.
Autorenporträt
Dr. Hains is currently an Adjunct Professor at the University of California, Berkeley. Previously he was an Assistant Professor of Neurology at the Yale University School of Medicine. He completed his graduate work at the University of Texas Medical Branch, and undergraduate training at Stetson University.